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(Currently in 2004 revised printing) a quick, transparent, thorough, and hugely relaxing method of medical microbiology, brimming with mnemonics, humor, precis charts and illustrations, from AIDS to "flesh-eating micro organism" to ebola, mad cow disorder, hantavirus, anthrax, smallpox, botulism, and so forth. very good Board evaluation.
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(Currently in 2004 revised printing) a short, transparent, thorough, and hugely stress-free method of scientific microbiology, brimming with mnemonics, humor, precis charts and illustrations, from AIDS to "flesh-eating micro organism" to ebola, mad cow disorder, hantavirus, anthrax, smallpox, botulism, and so forth. first-class Board overview.
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Extra resources for Clinical Microbiology Made Ridiculously Simple, Edition 3
A computerized tomogram (CT) scan reveals pockets of gas within the muscles and subcutaneous tissue. As the enzymes de42 CHAPTER 6. BACILLUS AND CLOSTRIDIUM (SPORE-FORMING RODS) Clostridium difficile must be considered as a possible cause. Samples of the stool can be sent to the laboratory for a Clostridium difficile toxin test. Toxin in the stool confirms the diagnosis. Treatment includes discontinuing the initial antibiotic and administering metronidazole or vancomycin by mouth. Both antibiotics kill Clostridium difficile and are not absorbed orally into the bloodstream.
Bacillus enjoys oxygen (so is aerobic), while Clostridium multiply in an anaerobic environment. In an air tight Closet, if you will! BACILLUS There are 2 pathogenic species of gram-positive, aerobic, spore-forming rods: Bacillus anthracis and Bacillus cereus. Bacillus anthracis causes the disease anthrax while Bacillus cereus causes gastroenteritis (food poisoning). Fig. 6-1. Anthony has Anthrax. This figure demonstrates how the Bacillus anthracis spores are contracted from contaminated products made of hides and goat hair.
Enterococcus now resistant to ampicillin and vancomycin! The enterococci are resistant to most of the drugs we use to kill gram positive bacteria. We usually treat enterococcal infections with ampicillin plus an aminoglycoside. However, many enterococcal strains are now resistant to both of these agents; in these cases we treat with vancomycin (see Fig. 16-17). S. The resistance property is carried on a gene that is transferable. Enterococci with this resistance gene alter their cell wall dipeptide d-alanine-d-alanine (the target for vancomycin, see page 141), changing it to d-alaninelactate.
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